Aortic plaque is when fatty deposits, calcium, and other substances collect along the inner lining of the aorta. Over time, these deposits can harden and narrow the artery, a condition known as atherosclerosis, which restricts blood flow to the heart and increases the risk of serious cardiovascular complications, such as:
- Blocked blood flow to your colon
- Blocked blood flow to your limbs
- Heart attack
- Blocked blood flow to your kidneys
- Blocked blood flow to your spleen
- Stroke or transient ischemic attack (TIA)
The biggest concern with plaque buildup in your aorta is the risk of an embolus, which is fragments of plaque that travel through your bloodstream until it gets stuck in a smaller artery, cutting off blood flow.
Plaque forms slowly over time, similar to how soap scum builds up inside a sink drain. But an embolus causes a sudden blockage—like accidentally dropping a toothpaste cap into the drain, instantly stopping the flow of water.
There are two types of emboli (blockages) that can result from aortic plaque:
- Thromboembolism: A blood clot that forms on the surface of the plaque. If a piece of this clot breaks off, it can travel through your bloodstream and cause a blockage.
- Atheroembolism: A fragment of plaque that breaks loose and moves through your bloodstream. This is caused when the plaque ruptures, releasing cholesterol crystals. Atheroembolisms are less common than thromboembolisms.
As aortic plaque does not result in any noticeable symptoms in its early stages, early detection and management are essential in reducing cardiovascular risks and preventing complications, such as stroke, heart attack, or heart failure.